CCHMC-eMERGE-Phase IIIA

Request Access

Variants of thymic stromal lymphopoietin and its receptor associate with eosinophilic esophagitis.

J. D. Sherrill, P. Gao, E. Stucke, et al.. (2010). The Journal of allergy and clinical immunology. Cited 261 times. https://doi.org/10.1016/j.jaci.2010.04.037

Eotaxin Triggers Eosmophil-Selective Chemotaxis and Calcium Flux via a Distinct Receptor and Induces Pulmonary Eosinophilia in the Presence of Interleukin 5 in Mice

M. Rothenberg, Robert T. Ownbey, P. Mehlhop, et al.. (1996). Molecular Medicine. Cited 202 times. https://doi.org/10.1007/BF03401631

Juvenile rheumatoid arthritis as a complex genetic trait.

D. Glass, E. Giannini. (1999). Arthritis and rheumatism. Cited 187 times. https://doi.org/10.1002/1529-0131(199911)42:11<2261::AID-ANR1>3.0.CO;2-P

Identification of a mouse eosinophil receptor for the CC chemokine eotaxin.

J. Gao, A. Sen, M. Kitaura, et al.. (1996). Biochemical and biophysical research communications. Cited 89 times. https://doi.org/10.1006/BBRC.1996.0955

Phenome-wide association study (PheWAS) in EMR-linked pediatric cohorts, genetically links PLCL1 to speech language development and IL5-IL13 to Eosinophilic Esophagitis

B. Namjou, Keith A. Marsolo, R. Caroll, et al.. (2014). Frontiers in Genetics. Cited 81 times. https://doi.org/10.3389/fgene.2014.00401

Genome-wide association analysis of juvenile idiopathic arthritis identifies a new susceptibility locus at chromosomal region 3q13.

S. Thompson, M. Marion, M. Sudman, et al.. (2012). Arthritis and rheumatism. Cited 68 times. https://doi.org/10.1002/art.34429

EMR-linked GWAS study: investigation of variation landscape of loci for body mass index in children

B. Namjou, M. Keddache, Keith A. Marsolo, et al.. (2013). Frontiers in Genetics. Cited 51 times. https://doi.org/10.3389/fgene.2013.00268
NCPI Dataset Catalog
Feedback & Support
v0.9.0-d9e5747